摘要：

Presents a study on the 4-kilodalton amyloid beta protein (betaAP), which is deposited as amyloid in the brains of patients with Alzheimer's disease. Derivation from a large protein, the amyloid beta protein precursor (BetaAPP); Cells normally producing and releasing soluble 4-kilodalton BetaAP that is essentially identical to the 4-kilodalton BetaAP deposited as insoluble amyloid fibrils in Alzheimer's disease.

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