The changing landscape of ischaemic brain injury mechanisms.
摘要：
Thrombolysis has become established as an acute treatment for human stroke. But despite multiple clinical trials, neuroprotective strategies have yet to be proved effective in humans. Here we discuss intrinsic tissue mechanisms of ischaemic brain injury, and present a perspective that broadening of therapeutic targeting beyond excitotoxicity and neuronal calcium overload will be desirable for developing the most effective neuroprotective therapies.
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关键词：
Humans Brain Ischemia Potassium Calcium Zinc Receptors, N-Methyl-D-Aspartate Apoptosis Thrombolytic Therapy Forecasting
DOI：
10.1038/399a007
被引量：
年份：
1999
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