Manipulation of the host actin cytoskeleton by Salmonella--all in the name of entry.
摘要：
The invasive pathogen Salmonella enterica has evolved sophisticated mechanisms to subvert the cytoskeletal machinery of its host. Following contact with the host cell, it delivers a distinct arsenal of effector proteins directly into the cytoplasm. These bacterial effectors coordinate transient actin rearrangements and alter vesicle trafficking to trigger invasion, without causing overt cellular damage. Recent studies have shed new light on the signaling mechanisms underlying this remarkable host–pathogen interface, in particular, highlighting the unique multi-functional role and temporal regulation of key bacterial effectors.
展开
关键词：
EPITHELIAL-CELLS ARP2/3 COMPLEX BACTERIAL INVASION RHO-GTPASES BINDING PROTEINS III SECRETION TYPHIMURIUM ACTIVATION NUCLEATION SIPA
DOI：
10.1016/j.mib.2004.09.001
被引量：
年份：
2005
Elsevier
国家科技图书文献中心
(权威机构)
Semantic Scholar
(全网免费下载)
Semantic Scholar
掌桥科研
查看更多
dx.doi.org
Oxford Univ Press
NCBI
Europe PMC
NCBI
ResearchGate
EBSCO
Citeseer
EBSCO
Citeseer
(全网免费下载)
msb.embopress.org
readcube.com
osti.gov
(全网免费下载)
plantphysiol.org
core.ac.uk
pubfacts.com
hw-f5-msb.highwire.org
mysciencework.com
deepdyve.com
pubget.com
www.socolar.com
tools.medicine.yale.edu
(全网免费下载)
molbiolcell.org
mendeley.com
通过文献互助平台发起求助，成功后即可免费获取论文全文。
相似文献
参考文献
引证文献
引用走势
辅助模式
引用
文献可以批量引用啦~
欢迎点我试用！
