The lpr and gld genes in systemic autoimmunity: life and death in the Fas lane
摘要：
The single gene lpr and gld models of spontaneous systemic autoimmunity have attracted much attention in recent years. Here, Philip Cohen and Robert Eisenberg describe the fascinating recent findings that the lpr and gld [corrected] phenotypes result from defects in the Fas gene and, perhaps, in the ligand for fas, respectively.
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关键词：
Animals Mice Mice, Mutant Strains B-Lymphocytes T-Lymphocyte Subsets Autoimmune Diseases Disease Models, Animal Lymphoproliferative Disorders Membrane Proteins Antigens, CD95
DOI：
10.1016/0167-5699(92)90066-G
被引量：
年份：
1992
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