Notch-dependent VEGFR3 upregulation allows angiogenesis without VEGF–VEGFR2 signalling
摘要：
DLL4–Notch signalling suppresses endothelial sprouting and angiogenic growth through crosstalk with the vascular endothelial growth factor (VEGF) pathway; VEGF receptor 2 has been thought to have a crucial role in this crosstalk, but now VEGF receptor 3 is shown to be the more important modulator.
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关键词：
Endothelial-Cells Kinase Inhibitors Tumor Angiogenesis Expression Network Formation Growth Dll4 Tip Cell-Formation Bevacizumab Activation
DOI：
10.1038/nature10908
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Springer
Nature
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